The concept of a miasm developed prior to an understanding of microbes as a cause of disease.

The word miasm was first used by Hippocrates as a “fault or a taint” in water or air as a cause for the transmission for certain diseases.

Miasms described by Hahnemann are infectious principles or agents that are passed from one generation to another. Transmission is vertical, from parent to child, not lateral as in contagion such as in measles. He described two broad categories of miasms: venereal and non-venereal. In the venereal category Hahnemann listed fig wart and chancre. In the non-venereal category he included psora or “itch” and tuberculinum. We now classify fig wort as Condyloma accuminata, a growth associated with the Human Papilloma Virus; chancre is due to Treponema pallidum or syphilis. Psora, obtained from fluid from a scabies cyst is associated with the Sarcoptes scabiei, the acarus mite; tuberculinum is associated with Mycobacteria.

There are other miasmatic agents than those described by Hahnemann. These include various bacteria, some viruses and fungi, and non-infectious agents like drugs, chemicals and biologicals. Any agent that can be identified in successive generations in which direct exposure can be ruled out should be considered a “miasm” from an electrodermal standpoint.

An example of vertical transmission is congenital syphilis. In this case one of the parents is infected with either a classical spirochete or or a variant cell wall deficient form (CWD) of it.

This commonly undetectable variant of Treponema pallidum may also cross the placenta to produce an attenuated form of syphilis, giving a clinical picture of a syphilitic miasm rather than a classical picture of congenital syphilis. I will discuss the concept of a polythetic class of bacteria below. In my database, the signal for syphilis is the most frequent and widespread of all the signals. Almost none of those tested had a history of being diagnosed with syphilis. One of the interesting features of syphilis is that it is very frequently difficult to see a spirochete in a microscopic slide from a chancre, the skin lesion of syphilis. The same is true for another spirochete, Borrella burgdorferi. It is rare to find a spirochete in the area of erythema (bull’s eye) or from draining lymph nodes in a case of Lyme disease. Morphologic changes occur in both spirochetes making their spiral configuration unrecognizable by microscopy. They become cell wall deficient.

There are three types of cells:

Cells that classically have an intact cell wall: coccus, bacillus or spirillum or filament

Cells that have complete or partial absence of a cell wall (CWD). They form large budding yeast-like forms, discharging cysts, budding or branching filamnents, balloons with sprouting rhizoids, syncitia or sheets.

Cells with no cell wall: protoplasts. These cells have a different DNA than the CWD’s without a cell wall.


Native bacteria and fungi have an intact cell walls but they may become cell wall deficient (CWD) when the cell wall is partially or completely removed.

Cell wall deficient forms are also known as L-Forms” (Lister Institute) named by Kleineberger-Nobel; “L-phase” or “Spheroplasts”. Most bacteria and many fungi can be induced to become cell wall deficient by changing their microenvironment, such as the oxygen content in which they are growing, temperature, pH, and electrolyte concentrations, types of amino acids and by bacteriocins (toxic molecules produced by some bacteria) and by exposure to chemical toxins including antibiotics.

Once bacteria or fungi are transformed into CWD’s their morphology, metabolism, and staining characteristics are changed. Even the pathogenicity of the organism may be changed.

In this form the organism may not be recognizable by the usual methods of detection. They may form slender branching filaments with buds of various sizes and locations or they may form sheets.

Colony appearance, sensitivity to penicillin, nutrient requirements and biochemical activities vary from species to species.

As mentioned before,two microbes that readily transform into cell wall deficient forms are the spirochetes Treponema pailidum and Borrelia burgdorferi, the causes for syphilis and Lyme borreliosis respectively. Instead of spirochetes being identified in a chancre of syphilis or in the skin or the lymph nodes of borreliosis, granular structures, mycelia or large bodies are found. With special techniques these unusual structures may be made to revert to their parental spirochete form.

Reversion can also take place naturally, but it is rarely a complete reversion.

The recognition that bacteria can be transformed into cell wall deficient forms and that they may revert to their parental forms implies that bacteria may have more than one morphologic form-they are pleomorphic .They undergo developmental changes and adapt to environmental changes Only in one stage is the organism monomorphic, a classic rod, coccus, or spirillum.

Each of the pleomorphic forms may be produce a disease that may or may not be exactly the same as that of the parent organism.1

Each pleomorphic cell wall deficient forms belong to a Polythetic Class, a category or class that is defined in terms of a broad set of criteria. One part of the set is neither necessary nor sufficient to define the entire set. Each member of the category must possess a certain minimal number of defining characteristics, but none of the features has to be found in each member of the category.

See a graphic illustration of a polythetic class in my journal entry on Taxonomy of viruses and other critters-fuzzy sets and hazy boundaries by van Ragenmortel.2 He illustrates the polythetic nature of viruses and demonstrates that viruses are more diverse than one would think by depending on morphology alone. This concept helped me to understand why we see changing electromagnetic changes in signals for species of microbes during sequential testing while signals for the genus of the organism remain the same. With the influenza virus there are “shifts” and “drifts in the type of flu that occurs from year to year implying a developmental or an environmental adaptation of this polythetic virus. See my log, An Overview of Influenza.


The following outline should help you to recognize the major miasms. Further investigation is needed to correlate other microbial and non infectious agents in order to classify them as miasmatic.

Psoric Miasm:

Overview: functional disturbances like headache, nausea, and discomfort from noise, light, and odors.

Personality: Highs and lows, struggles with outside world in times of stress, lack of confidence, constant anxiety about the future, fear, like he can’t do it, insecurity but has hope, mentally alert.

Physical: Itching, burning, inflammation leading to congestion – philosopher, selfish, restless, weak, fears.

Skin effects: Dirty, dry, itching without pus or discharge, burning, scaly eruptions, eczema, cracks in hands and feet, sweat profuse worse during sleep, offensive.

Pain: Neurological type, sore, bruised, better rest , worse motion.

Clinical diseases: Acidity, burning, cancer, sarcomas, constipation, epilepsy, flatulence, hoarseness, itching of skin, leprosy, burning of spinal cord, watery discharge from nose and eyes with burning.

Electrodermal detection sites: Lymphatic degeneration OR 1-1

Electrodermal signals: Psorrinum


Sycotic Miasm

Overview: Hypersensitive (hypertrophic) response to something specific, like tumors, allergies, keloids. Deficient feeling gives rise to an increased attempt to repair the fault.

Personality: Secretive, hides his weakness, tense, constantly covering up situations, fixed habits, suspicious, jealous, forgetful.

Physical: Over production, growth-like warts, condylomata, fibrous tissue, attacks internal organs, pelvis, and sexual organs.

Skin effects: Warts, moles, unnatural thickening of skin, herpes, scars, nails are thick, irregular & corrugated, oily skin with oozing, disturbed pigment in patches.

Pain: Joint pains, stitching, pulsating, wandering, rheumatic pains are worse cold, damp, better motion.

Clinical diseases: Abortion, acne without pus, anemia, cough, colic, pelvic disease, piles, prostatitis, nephritis, dysmenorrhea, warts, urinary ailments, overgrowth of tissue anywhere in the body, benign enlargements like cartilage in rheumatoid arthritis.

Electrodermal detection sites: Lymphatic degeneration OR 1-1; Skin Detection site SK 1-3; Penis/Vagina BL 51

Electrodermal signals: Medorrhinum; Neiserria gonorrhea; Condyloma accuminata; Human papilloma virus.

Syphilitic Miasm

Overview: Destructive disorder everywhere, ulceration, fissures, deformities, suicidal, depression, diminished memory.

Physical: Gangrenous ulcerations.

Personality: Strong, pessimistic, gives-up easily, destructive behavior, risk taking, sudden impulsive violence directed at oneself or others, distorted rigid ideas. Mental paralysis, mentally dull, stubborn, suicidal and homicidal; alcohol abuse

Skin effects: Ulcers, boils, offensive discharge of fluids and pus, slow to heal, hair loss, worse from heat of bed, spoon shaped thin nails that tear easily, putrid gangrene.

Pain: Bone pains that are tearing, bursting, burning; lancinating or shooting pain in the extremities, especially at night, with the symptom of “dread of night”

Clinical diseases: Putrid discharges, blindness, ulcerations in veins and bones, carcinoma (has not been ruled out), fistulas, gangrene, hyperextension (Charcot’s Joint), bone marrow inflammation, insanity due to depression, leucorrhoea, skin disease, ulcerated sore throat, history of abortions, sterility, premature death in infants, ischemic heart disease, ulcerations of blood vessels leading to hypertension, heart attack and stroke, suicidal deaths, insanity, ulcers of ear, nose, urinary organs andmouth.

Electrodermal detection sites: Lymphatic degeneration OR 1-1

Electrodermal signals: Treponema palliduM

Tubercular Miasm

Overview: Never satisfied, intolerant, changes everything, does harmful thing to ones self.

Personality: Changing symptoms, logy, vague, weak, moves location frequently, careless, “problem child”, harmful cravings.

Physical: Recurring boils with pus and fever, does not heal fast. Worse from warmth of bed; better cold; nails with white spots.

Pain: Aching pain in knees with swelling

Clinical diseases: bedwetting, cancer, carious teeth, destruction of bone marrow, diabetes, dry cough (barking), eczema, emaciation, epilepsy, extreme fatigue, weakness, glands enlarged, tonsils, influenza, insanity, obstruction of intestines, malaria, insomnia, nocturnal perspiration, palpitation, profuse hemorrhage of any orifice, pneumonia, ring worm, nasal coryza.

Electrodermal detection sites: Lymphatic degeneration OR 1-1

Electrodermal signals: Tubercular or non-tubercular mycobacteria


Electromagnetic imprinted information in biological water of the parent is another way of transferring information from one generation to the next.

There are two types of imprinting, one is biological, and the other is computer based. In the first case, organisms living in a host have electromagnetic fields (auras) that extend into the surrounding host structures producing an electromagnetic field effect in those structures.

A good example is the effect that Enterobius vermicularis (pinworm) has on its host. This helminth produces intense pruritis ani (anal itching) in children. Generally, in adolescence and in adulthood, the pinworm is no longer present but, in some instances, the pruritis persists. This implies that there was a residual effect from the pinworm that causes itching in the tissue adjacent to its nesting site. Since all tissue contains water, there is a strong probability that the electromagnetic field of the pinworm has been imprinted in the water of the mucosa, the fibrous connective tissue or in the nerves of the surrounding area. This may be an electromagnetically induced cause of the pruritis even though the pinworm is no longer there. The possibility that the pruritis is an electro magnetic effect is strengthened by the relief of the symptoms of pruritis after an electromagnetically induced solution of Enterobius vermicularis is applied to the tongue.

A homeopathic remedy made from Enterobius vermicularis is another way to treat this type of pruritis. It is called isopathy because it is made from the helminth Enterobius vermicularis. This isonosode has a signal equal to Enterobius vermicularis, the cause of the pruritis. The homeopathic iso-nosode is an equilium of Enterobius vermicularis.

The electromagnetically imprinted signal acts like the signal for native Enterobius vermicularis. Equiliums are not the same as Hahnemann’s similimums. Similimums are expressions of the totality of symptoms that are similar to the disease for which it was being used to treat. Electromagnetically the signal for a similimum signal can also be detected at sites expressed by the disease.

Occasionally, miasms can be detected at TW 20, the Hypothamic Detection Site. Since they have a conductance effect at the Hypothalamus Site, they exhibit a constitutional effects.

Typically, Constitutional signals are found at TW 20 although I had a case of thyroid carcinoma that resolved with Natrum Muriaticum when tested at TW 20 the hypothalamic gland site and at the Thyroid Gland Site ST 10. She had been humiliated and falsely accused of cheating in school as a child and had low personal esteem and depression ever since. The electromagnetic signal for Natrum Muriaticum matched the Constitutional state of  Natrum Muriaticum  which is homeopathically used for depression and low personal esteem.

Miasms have been viewed as microbial in nature but, in my view, they may be non-microbial too. Drugs and other chemicals can be detected vertically or trans-generationally by electrodermal means.

As an example, it is not uncommon to identify a code for cocaine in children whose parents used cocaine, even though the children were not exposed.

Codes for cocaine and other recreational drugs are located at the same sites as their parents, GB 17, the Reticular Formation Site; NV 1—1, the Neurotransmitter Site, and SV 52, the Acetylcholine Receptor Site. This transfer of information from parent to the child is a biological transfer, most likely in body water of seminal fluid or vaginal fluid, spermatozoa or ova.

List of Miasm Codes

This list includes only those miasms reported in my text An Electrodermal Analysis of Biological Conductance. Encrypted codes for additional miasms can be found in most electrodermal detection devices.

Nosodes are pathological specimens digitally converted to electrodermal codes.

Bacillinum: a homeopathic nosode made by J. Compton Burnett, from the lung of a patient with tuberculosis.

Carcinosinum: a homeopathic nosode made from a combined mixture of malignant tumors including those originating in the breast, stomach, bladder, intestine and lung.

Medorrhinum: a nosode made from Neisseria gonorrhea, the bacterial cause of gonorrhea.

Psorinum: a nosode made from a clear or purulent vesicle of the Arcaris scabiei itch mite.

Sarcominum: a nosode made from a sarcoma.

Scirrhinum: a nosode made from scirrhous carcinoma of the breast. Streptococcinum: a nosode made from a bloody gingival sac containing Streptococcus proteiformis, a variant of Streptococcus faecalls. Syphilinum (Leuesinum): a nosode made from the serous fluid of a syphilitic chancre.

Tuberculinum: nosode made from a liquid culture of Mycobacterium tuberculosis.

Tuberculinum Burnett: a nosode made from an isolate from the tissue of a lung with tuberculosis by J. Compton Burnett.

Tuberculinum Denys: a nosode made by Denys of Louvain in 1896 that contained a thermolabile “toxalbumin”.

Tuberculinum Kent: a nosode made from an isolate of tuberulosis by Kent. Tuberculinum Koch: a nosode of Mycobacterium tuberculosis isolated from a liquid culture by Koch.

Tuberculinum Residuum: a nosode made from Mycobacterium residuum, a soil bacterium.

Tuberculinum Rosenbach: a tuberculosis nosode characterized by Rosenbach.


1. Cell Wall Deficient forms: Stealth Pathogens/ Lida Mattman CRC Press Inc. 2nd edition 1992.

2. Virus Taxonomy.7th Report of the International Committee on Virus Taxonomy, van Regenmortel ed. Academic Press, San Diego 2000 page 5

3. Miasms by David Little www.simillimum.com

4. Materia Medica of the Nosodes H.C. Allen 1908; B.Jain Publishers, New Delhi, Reprinted 1988.

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